Group of hemorrhagic disorders in which the VON WILLEBRAND FACTOR is either quantitatively or qualitatively abnormal. They are usually inherited as an autosomal dominant trait though rare kindreds are autosomal recessive. Symptoms vary depending on severity and disease type but may include prolonged bleeding time, deficiency of factor VIII, and impaired platelet adhesion
Group of hemorrhagic disorders in which the VON WILLEBRAND FACTOR is either quantitatively or qualitatively abnormal. They are usually inherited as an autosomal dominant trait though rare kindreds are autosomal recessive. Symptoms vary depending on severity and disease type but may include prolonged bleeding time, deficiency of factor VIII, and impaired platelet adhesion
Vascular resistance. : Onde di polso : Come l'emodinamica vascolare determina la pressione arteriosa / Paolo Salvi ; Presentazione a cura di Alberto Zanchetti
Constriction of arteries in the SKULL due to sudden, sharp, and often persistent smooth muscle contraction in blood vessels. Intracranial vasospasm results in reduced vessel lumen caliber, restricted blood flow to the brain, and BRAIN ISCHEMIA that may lead to hypoxic-ischemic brain injury (HYPOXIA-ISCHEMIA, BRAIN)
Constriction of arteries in the SKULL due to sudden, sharp, and often persistent smooth muscle contraction in blood vessels. Intracranial vasospasm results in reduced vessel lumen caliber, restricted blood flow to the brain, and BRAIN ISCHEMIA that may lead to hypoxic-ischemic brain injury (HYPOXIA-ISCHEMIA, BRAIN)
Deposition of calcium into the blood vessel structures. Excessive calcification of the vessels are associated with ATHEROSCLEROTIC PLAQUES formation particularly after MYOCARDIAL INFARCTION (see MONCKEBERG MEDIAL CALCIFIC SCLEROSIS) and chronic kidney diseases which in turn increase VASCULAR STIFFNESS
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Vascular Stiffness : Advances in arterial stiffness research / Becky Chavez, editor
2016
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Vascular Stiffness -- physiology : Early vascular aging (EVA) : new directions in cardiovascular protection / edited by Peter M. Nilsson, MD, PhD, Michael H. Olson, MD, PhD, Stéphane Laurent, MD, PhD
Vascular Surgical Procedures -- methods -- Case Reports : Clinical scenarios in vascular surgery / editors, Gilbert R. Upchurch, Jr, MD, Chief of Vascular and Endovascular Surgery, William H. Muller, Jr Professor of Surgery, Professor of Molecular Physiology and Biological Physics, University of Virginia, Charlottesville, Virginia, Peter K. Henke, MD, Leland Ira Doan Professor of Surgery, Chief, Vascular Surgery AAVA, University of Michigan, Ann Arbor, Michigan
Vascular Surgical Procedures -- trends : Modern trends in vascular surgery : ischemic extremities : new findings & treatment / [edited by] Heron E. Rodriguez, William H. Pearce, James S.T. Yao
Vascular system. : Peptides in energy balance and obesity / edited by Gema Frühbeck
2009
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Vascular System Injuries -- prevention & control : Vascular challenges in skull base surgery Paul A. Gardner, MD, Professor and Peter J. Jannetta Endowed Chair, Department of Neurological Surgery, University of Pittsburgh School of Medicine; Co-Director, Center for Cranial Base Surgery, University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania, USA, Carl H. Snyderman, MD, MBA, Professor, Department of Otolaryngology, University of Pittsburgh School of Medicine; Co-Director, Center for Cranial Base Surgery, University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania, USA, Brian T. Jankowitz, MD, Associate Professor, Director, Cerebrovascular Surgery, Department of Neurological Surgery, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania, USA
Widespread necrotizing angiitis with granulomas. Pulmonary involvement is frequent. Asthma or other respiratory infection may precede evidence of vasculitis. Eosinophilia and lung involvement differentiate this disease from POLYARTERITIS NODOSA
Widespread necrotizing angiitis with granulomas. Pulmonary involvement is frequent. Asthma or other respiratory infection may precede evidence of vasculitis. Eosinophilia and lung involvement differentiate this disease from POLYARTERITIS NODOSA
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Vasculitis -- Diagnosis : Vasculitis in clinical practice / Richard A. Watts, David G.I. Scott, Chetan Mukhtyar
Widespread necrotizing angiitis with granulomas. Pulmonary involvement is frequent. Asthma or other respiratory infection may precede evidence of vasculitis. Eosinophilia and lung involvement differentiate this disease from POLYARTERITIS NODOSA
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Vasculitis -- Handbooks, manuals, etc : The heart in systemic autoimmune diseases / edited by Fabiola Atzeni, Andrea Doria, Michael Nurmohamed, Paolo Pauletto
A systemic non-thrombocytopenic purpura caused by HYPERSENSITIVITY VASCULITIS and deposition of IGA-containing IMMUNE COMPLEXES within the blood vessels throughout the body, including those in the kidney (KIDNEY GLOMERULUS). Clinical symptoms include URTICARIA; ERYTHEMA; ARTHRITIS; GASTROINTESTINAL HEMORRHAGE; and renal involvement. Most cases are seen in children after acute upper respiratory infections
