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Mark   Year Entries
Spinks, Bryan D : The serious business of worship : essays in honor of Bryan D. Spinks / edited by Melanie C. Ross and Simon Jones  2012 1
Spinks, Don : Exit wounds : one Australian's war on terror / John Cantwell with Greg Bearup  2012 1
Spinks, Michael : Tony Brown's journal. Can Lewis and Spinks knock out apartheid? / directed by Bob Morris  1986 1
Spinmodell : Structure in complex networks / J. Reichardt  2009 1
Spinnen   5
Spinnen Motiv : Spider / Katarzyna and Sergiusz Michalski  2010 1
Spinnenaffe : Faces in the forest : the endangered muriqui monkeys of Brazil / Karen B. Strier  1992 1
Spinnennetz.   2
Spinnentiere. : Web of nature : Martin Lister (1639-1712), the first arachnologist / by Anna Marie Roos  2011 1
 

Spinner dolphin -- See Stenella longirostris


  1
 

Spinner porpoise -- See Stenella longirostris


  1
 

Spinnet (Piano) -- See Piano


  1
Spinney, George F   2
  Spinning -- 8 Related Subjects   8
Spinning.   20
 

Spinning (Aerodynamics) -- See Spin (Aerodynamics)


  1
Spinning -- Australia. : The behaviour of doggy and normal wool in the worsted spinning using the amber superdraft method / by T. Carter, W.R. Lang and R.B. Sweetten  1961 1
 

Spinning (Fishing) -- See Spin fishing


  1
Spinning -- History   3
Spinning in art. : Late antique images of the Virgin Annunciate spinning : allotting the scarlet and the purple / by Catherine Gines Taylor  2018 1
 

Spinning machinery -- See Also the narrower term Spinning-wheel


  1
Spinning machinery.   5
Spinning machinery -- Technological innovations. : Scope: Things That Spin  2009 1
Spinning -- Management : A practical guide to quality management in spinning / B. Purushothama  2011 1
spinning (metalworking) : Design for manufacturability handbook James G. Bralla, editor  2000 1
Spinning -- New Zealand -- History : The loving stitch : a history of knitting and spinning in New Zealand  1998 1
Spinning -- Periodicals. : The Australian hand weaver and spinner    1
Spinning -- Quality control   2
 

Spinning, Rope -- See Trick roping


  1
 

Spinning Sensation -- See Vertigo


An illusion of movement, either of the external world revolving around the individual or of the individual revolving in space. Vertigo may be associated with disorders of the inner ear (EAR, INNER); VESTIBULAR NERVE; BRAINSTEM; or CEREBRAL CORTEX. Lesions in the TEMPORAL LOBE and PARIETAL LOBE may be associated with FOCAL SEIZURES that may feature vertigo as an ictal manifestation. (From Adams et al., Principles of Neurology, 6th ed, pp300-1) 		  1
 

Spinning Sensations -- See Vertigo


An illusion of movement, either of the external world revolving around the individual or of the individual revolving in space. Vertigo may be associated with disorders of the inner ear (EAR, INNER); VESTIBULAR NERVE; BRAINSTEM; or CEREBRAL CORTEX. Lesions in the TEMPORAL LOBE and PARIETAL LOBE may be associated with FOCAL SEIZURES that may feature vertigo as an ictal manifestation. (From Adams et al., Principles of Neurology, 6th ed, pp300-1) 		  1
Spinning -- Texas -- History -- 19th century : Hands to the spindle : Texas women and home textile production, 1822-1880 / by Paula Mitchell Marks ; illustrated by Walle Conoly  1996 1
 

Spinning tops -- See Tops


  1
Spinning (Trademark) : Riding into the future : a financial examination of SoulCycle and the indoor cycling studio trend / Marion E. Hambrick  2017 1
Spinning-wheel.   6
Spinning-wheel -- Teaching aids : Spinning-wheel [realia]    1
 

Spinocerebellar Ataxia -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 1 -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 1s -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 2 -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 2s -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 3 Protein -- See Ataxin-3


A deubiquitinating enzyme of the ATAXINS family. It functions in protein homeostasis, GENETIC TRANSCRIPTION; CYTOSKELETON regulation, and MYOGENESIS. CAG TRINUCLEOTIDE REPEAT EXPANSION in the Ataxin-3 gene coding region is associated with spinocerebellar ataxia-3 (MACHADO-JOSEPH DISEASE) 		  1
 

Spinocerebellar Ataxia 4 -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 4s -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 5 -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 5s -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 6 -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 6s -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 7 -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
 

Spinocerebellar Ataxia 7s -- See Spinocerebellar Ataxias


A group of dominantly inherited, predominately late-onset, cerebellar ataxias which have been divided into multiple subtypes based on clinical features and genetic mapping. Progressive ataxia is a central feature of these conditions, and in certain subtypes POLYNEUROPATHY; DYSARTHRIA; visual loss; and other disorders may develop. (From Joynt, Clinical Neurology, 1997, Ch65, pp 12-17; J Neuropathol Exp Neurol 1998 Jun;57(6):531-43) 		  1
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