| Description |
1 online resource |
| Contents |
Machine generated contents note: Genetics, -- Diet, -- Hormones, -- Stress, -- Comedones (plugs in pores), -- Blemishes-a brief catalogue, -- Nodules, -- Scars and sinuses, -- Support, -- Nomenclature, -- The three acnes and grading, -- Acne vulgaris, -- Acne rosacea, -- Acne inversa (hidradenitis suppurativa), -- Grading the three acnes, -- Acne vulgaris, -- Acne rosacea, -- Acne inversa (hidradenitis suppurativa), -- 1.1. Acne vulgaris, -- 1.1.1. Terminology, -- 1.1.2. The starting point, -- 1.2. Acne rosacea, -- 1.2.1. The "pimply" part, -- 1.2.2. The "redness" part, -- 1.2.3. The third part, the firm fibrosis, -- 1.2.4. Part four-ocular rosacea, -- 1.2.5. Putting it all together, -- 1.2.6. The inflammatory epiphenomena in acne rosacea, -- 1.2.7. The "acne rosacea" versus "rosacea" controversy, -- 1.2.8. Summary, -- 1.3. Acne inversa (formerly hidradenitis suppurativa), -- 1.3.1. Before the rupture, where and why?, -- 1.3.2. After the rupture, what next?, -- 1.3.3. So what invaders are important in acne inversa?, -- 1.3.4. What makes this disease behave so much worse than acne vulgaris?, -- 1.3.5. So what can one possibly do to settle down all this inflammation?, -- 1.3.6. So how do you get rid of all this material?, -- 1.3.7. What does the future offer?, -- 1.4. The psychology of acne, -- 1.4.1. Acne as a stress, -- 1.4.2. Acne and self-image, -- 1.4.3. Isotretinoin therapy and the psyche, -- 1.4.4. The isotretinoin-depression question, -- 1.4.5. Isotretinoin in perspective, -- 2.1. Anatomy, -- 2.2. Genetics, -- 2.2.1. Acne vulgaris, -- 2.2.2. Acne rosacea, -- 2.2.3. Acne inversa/hidradenitis suppurativa (AI/HS), -- 2.2.4. The Scottish twins, -- 2.3. Epigenetics, -- 2.3.1. The farmer's boys, -- 2.4. Embryology, -- 2.5. Histology, -- 2.5.1. Onwards and downwards, -- 2.5.2. What is going on inside the FPSU?, -- 2.6. Physiology, -- 2.6.1. Hair first, -- 2.6.2. Oil second, -- 2.6.3. Last but definitely not least: the follicle, -- 2.6.4. Looking deeper, -- 2.7. Biochemistry, -- 2.8. Hormones, enzymes, receptors, and the intracrine system, -- 2.8.1. The intracrine system, -- 2.9. FoxO1 and mTORC1, -- 2.9.1. The next step, -- 2.9.2. The broad view, -- 3.1. Acne vulgaris, -- 3.2. Acne rosacea, -- 3.3. Acne inversa/hidradenitis suppurativa (AI/HS), -- 3.4. Other variants, -- 3.4.1. Malassezia folliculitis, -- 3.4.2. Eosinophilic pustular folliculitis (Ofuji's disease), -- 3.4.3. Dissecting terminal folliculitis, -- 3.4.4. Acne keloidalis, -- 3.4.5. Epidermal growth factor receptor (EGFR) inhibitor eruption, -- 3.4.6. Acne excoriee des jeunes files, -- 4.1. The endogenous hormones, -- 4.1.1. Androgens and their sources, -- 4.1.2. Estrogens and their sources, -- 4.1.3. Progesterone and the progesteroids, -- 4.1.4. Insulin, -- 4.1.5. Growth hormone and insulin-like growth factor-1, -- 4.2. The exogenous hormones, -- 4.2.1. Anabolic steroids, -- 4.2.1.1. Mothers' milk, -- 4.2.1.2. Muscle makers, -- 4.2.2. Oral contraceptive hormones, -- 4.2.2.1. Oral estrogens, -- 4.2.2.2. Oral progestins, -- 4.2.2.3. Extended cycles, -- 4.2.3. Other exogenous birth control hormones, -- 4.2.3.1. Implants, -- 4.2.3.2. Intrauterine devices, -- 4.2.3.3. Intravaginal devices, -- 4.2.3.4. Topicals: the patches, -- 4.2.3.5. Intramuscular (depot) injections, -- 4.2.4. Dietary sources of hormones, -- 4.2.4.1. The impact of diet on acne, -- 4.2.4.1.1. The ice cream salesman's son, -- 4.2.4.1.2. Reproductive hormones, -- 4.2.4.1.3. Insulin, -- 4.2.4.1.4. Insulin-like growth factor 1 (IGF-1), -- 4.2.4.1.5. Growth factors and androgens combined, -- 4.2.4.1.6. Dairy intolerance, -- 4.2.4.2. Carbohydrate load versus dairy load, -- 5.1. Chemicals and medications, -- 5.2. Endocrine imitators and disruptors, -- 5.2.1. Environmental contamination, -- 5.3. Foods, -- 5.3.1. Iodine and bromine, -- 5.3.2. Chocolate, -- 5.3.3. Casein and whey, -- 5.4. Photodamage, glycation, and the acne and aging processes, -- 5.5. Smoking and nicotine, -- 6.1. Propionibacterium acnes (P. acnes), -- 6.1.1. Normal role of P. acnes, -- 6.1.2. Pathogenic role of P |
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Acnes, -- 6.2. Malassezia species, -- 6.2.1. Normal role, -- 6.2.2. Immunogenicity, -- 6.2.3. Pruritogenicity, -- 6.2.4. Malassezia in the acnes, -- 6.3. Staph, Strep, and Gram-negative organisms, -- 6.4. Demodex, -- 6.5. Vellus hairs, -- 7.1. Innate immunity, -- 7.2. Adaptive (acquired) immunity, -- 7.3. Inflammation as the primary acnegen, -- 7.4. Mediators, cellular and humoral, and neuroimmunology, -- 7.5. Allergy (shared antigens), -- 7.6. Inflammation, pigment, and PIH, -- 7.7. Inflammation and scarring, -- 8.1. Prevention, -- 8.2. General principles of management, -- 8.3. Diet, -- 8.3.1. Dairy, -- 8.3.1.1. The deli-planning heiress, -- 8.3.1.2. The pharmaceutical executive, -- 8.3.2. Carbohydrates, glycemic load, and hyperinsulinemia, -- 8.3.3. The paleolithic diet, -- 8.3.4. High-fructose corn syrup (HFCS), -- 8.3.5. Metformin, -- 8.3.6. Synthesis and summary, -- 8.4.Comedolytics and other topicals, -- 8.4.1. Standard topical comedolytics, -- 8.4.1.1. Retinoids, -- 8.4.1.2. Benzoyl peroxide, -- 8.4.1.3. Salicylic acid, -- 8.4.1.4. Alpha and beta-hydroxy adds, -- 8.4.2. Unclassified topicals, -- 8.4.2.1. Azelaic acid, -- 8.4.2.2. Sulfur, -- 8.4.2.3. Zinc compounds, -- 8.4.2.4. Resorcinol, -- 8.4.3. Systemic comedolytics, -- 8.4.3.1. Vitamin A, -- 8.4.3.2. Isotretinoin, -- 8.4.3.2.1. Teratogenicity, -- 8.4.3.2.2. Contraception, -- 8.4.3.2.3. Inflammatory bowel disease, -- 8.4.3.2.4. Depression, -- 8.4.3.2.5. Other side effects, -- 8.4.3.2.6. The convict who looked like Chief, -- 8.4.3.3. Acitretin, -- 8.4.3.4. Summary, -- 8.5. Anti-inflammatories and antimicrobials, -- 8.5.1. Antibiotics as anti-inflammatories, -- 8.5.1.1. In acne vulgaris, -- 8.5.1.2. In acne rosacea, -- 8.5.1.3. In acne inversa, -- 8.5.1.4. In dissecting terminal folliculitis (DTP) and acne keloidalis, -- 8.5.2. Antibiotics as antibiotics, -- 8.5.3. Ketoconazole, ivermectin, and crotamiton, -- 8.5.3.1. In acne vulgaris, -- 8.5.3.2. In acne rosacea, -- 8.5.3.3. In acne inversa/hidradenitis suppurativa and dissecting folliculitis and cellulitis, -- 8.5.4. Steroids, -- 8.5.4.1. The Marine, -- 8.5.5. Nonsteroidal anti-inflammatory drugs (NSAIDs) and biologics, -- 8.5.6. Phototherapy, -- 8.5.7. Post-inflammatory hyperpigmentation, -- 8.5.7.1. Prognosis, -- 8.6. Hormone manipulations and therapy, -- 8.6.1. Birth control pill selection, -- 8.6.1.1. Estrogens, -- 8.6.1.1.1. Warnings, -- 8.6.1.2. Progestins, -- 8.6.2. Androgen receptor blockade, -- 8.6.2.1. Spironolactone, -- 8.6.2.2. Cyproterone acetate, -- 8.6.2.3. Flutamide, -- 8.6.2.4. Drospirenone, -- 8.6.2.5. Topical androgen blockers, -- 8.6.3. Dihydrotestosterone minimization, -- 8.6.3.1. Finasteride, -- 8.6.3.2. Dutasteride, -- 8.6.3.3. Diet, -- 8.6.4. Phototherapy-hormone interactions, -- 8.7. Surgery, -- 8.7.1. Acne vulgaris, -- 8.7.1.1. Acne surgery for patients, -- 8.7.1.2. Acne surgery for physicians, -- 8.7.2. Acne rosacea, -- 8.7.3. Acne inversa/hidradenitis suppurativa, -- 8.7.3.1. Mini-unroofing by punch biopsy, -- 8.7.3.2. Unroofing, -- 8.7.3.2.1. The Trucker, -- 8.7.3.3. Wide surgical excision, -- 8.7.3.4. Healing options, -- 8.7.3.4.1. Primary closure, -- 8.7.3.4.2. Secondary intention, -- 8.7.3.4.3. Split-thickness mesh grafting, -- 8.8. Lights and lasers, -- 8.8.1. Light and other radiation in acne, -- 8.8.1.1. Radiation's targets, -- 8.8.1.2. Light as a practical acne therapy, -- 8.8.2. Lasers, -- 9.1. Epidemiology, -- 9.2. Pathogenesis, -- 9.3. Team up with Mother Nature, -- 9.4. Targeting therapy, -- 9.4.1. Clinical manifestations, -- 9.4.2. Pathology, -- 9.4.3. Diagnostic evaluation, -- 9.4.4. Overview and general approach to treatment, -- 9.4.5. Milk and pregnancy, -- 9.4.6. Active therapy, -- 9.4.6.1. Avoidance of harm, -- 9.4.6.2. Lesion-directed therapy, -- 9.4.6.3. Nonprescription topicals, -- 9.4.6.4. Antimicrobials, -- 9.4.6.5.Combination topicals, -- 9.4.6.6. Anti-inflammatories, -- 9.4.6.7. Hormone blockers, -- 9.4.6.8. Procedural therapies, -- 9.5. Discussion, -- 9.6. Summary and conclusion, -- 10.1. Lifestyle choices and the acnes, -- 10.1.1. The "processed cheese queen", -- 10.2. Therapeutic choices and the acnes, -- 10.2.1. Acne vulgaris, -- 10.2.2. Acne rosacea, -- 10.2.3. Acne inversa/hidradenitis suppurativa, -- 10.3. Conclusion, -- 11.1. Appendix A: the rosacea "classification and staging" controversy, -- 11.2. Appendix B: the dairy versus carbohydrate controversy, -- 12.1. Acne, -- 12.2. The "zero-dairy" diet, -- 12.3. The risks and benefits of isotretinoin, -- 12.4. The Paleo diet, -- 12.5. Acne inversa/Hidradenitis suppurativa (AI/HS), -- 12.6. Yasmin/Ocella/Zarah or Yaz/Gianvi extended cycle for acne therapy |
| Summary |
"Acne is a common ailment for teenagers but can persist well into middle age. Although the formation of comedones in hair follicles is quite well understood, the actual causes initiating the process are less well so. Many theories have been forwarded, from hormones through cleanliness to diet, none of which fully explain the condition. Indeed the variation in presentation and classification (there are three acnes) can make precise diagnosis troublesome. It is likely the causes are multifactorial, and evidence is accruing for all the above contributors. Many books have focused on the straightforward pathophysiological processes of hormones and cleanliness but have neglected the contribution of diet as a possible guide to prevention and other medical approaches to treatment. This book will take more catholic view, with a practical focus on all the possible modes of treatment based on current pathophysiological knowledge. The aim of the book is to assemble the pieces of the jigsaw puzzle so that the resulting picture will be comprehensible to physicians and other caregivers, and identify what is known and what needs more investigation"--Provided by publisher |
| Bibliography |
Includes bibliographical references and index |
| Notes |
English |
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Print version record and CIP data provided by publisher |
| Subject |
Acne.
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Acne -- Treatment
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Suppuration.
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Sweat glands -- Diseases.
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Bacterial diseases.
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Infection.
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Diseases.
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Skin -- Diseases.
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Rosacea.
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Acne Vulgaris
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Acneiform Eruptions
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Hidradenitis
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Suppuration
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Skin Diseases, Infectious
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Facial Dermatoses
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Skin and Connective Tissue Diseases
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Sweat Gland Diseases
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Bacterial Infections
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Infections
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Bacterial Infections and Mycoses
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Disease
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Skin Diseases, Bacterial
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Sebaceous Gland Diseases
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Skin Diseases
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Hidradenitis Suppurativa
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Rosacea
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disease.
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illness.
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HEALTH & FITNESS -- Diseases -- General.
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MEDICAL -- Clinical Medicine.
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MEDICAL -- Diseases.
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MEDICAL -- Evidence-Based Medicine.
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MEDICAL -- Internal Medicine.
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Acne.
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Acne -- Treatment.
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| Form |
Electronic book
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| LC no. |
2014032981 |
| ISBN |
9781118272367 |
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1118272366 |
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9781118272381 |
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1118272382 |
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9781118272343 |
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111827234X |
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