Immune system -- Infections : Infections in systemic autoimmune diseases : risk factors and management / edited by Fabiola Atzeni, James B. Galloway, Juan J. Gomez-Reino, Massimo Galli
2020
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Immune System -- innervation : Cell communication in nervous and immune system / [edited by] Eckart D. Gundelfinger, Constanze I. Seidenbecher, Burkhart Schraven
Immune system -- Nurses' Instruction : The human immune system : the immune response / executive in charge of production, Tina Armstrong ; executive producer, Rand Rodriguez ; designer and director, Jon Frank ; writer, John Shannon
Immune system -- Phenomena -- Nurses' Instruction : The human immune system : the immune response / executive in charge of production, Tina Armstrong ; executive producer, Rand Rodriguez ; designer and director, Jon Frank ; writer, John Shannon
The body's defense mechanism against foreign organisms or substances and deviant native cells. It includes the humoral immune response and the cell-mediated response and consists of a complex of interrelated cellular, molecular, and genetic components
Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
Thrombocytopenia occurring in the absence of toxic exposure or a disease associated with decreased platelets. It is mediated by immune mechanisms, in most cases IMMUNOGLOBULIN G autoantibodies which attach to platelets and subsequently undergo destruction by macrophages. The disease is seen in acute (affecting children) and chronic (adult) forms
