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Politische Institution -- Glück. : Happiness and economics : how the economy and institutions affect well-being / Bruno S. Frey and Alois Stutzer  2002 1
 

Gluck, R. Ch. W. von (Christoph Willibald), Ritter von, 1714-1787 -- See Gluck, Christoph Willibald, Ritter von, 1714-1787


  1
Glück -- Wirtschaft. : Happiness and economics : how the economy and institutions affect well-being / Bruno S. Frey and Alois Stutzer  2002 1
 

Glückel, of Hameln, 1646-1724 -- See Glueckel, of Hameln, 1646-1724


  1
 

Glückel, von Hameln, 1646-1724 -- See Glueckel, of Hameln, 1646-1724


  1
Gluckman, David : Lumkani : rapid detection of fires in informal settlements / Chris van der Hoven, Stephanie Townsend  2018 1
Gluckman, Max, 1911-1975.   2
 

Gluckmann, Herman Max, 1911-1975 -- See Gluckman, Max, 1911-1975


  1
Glückseligkeit : Teaching Happiness and Well-Being in Schools : Learning to Ride Elephants / Ian Morris  2009 1
Glücksspiel   9
Glücksspielindustrie : Intimate economies of development : mobility, sexuality and health in Asia / Chris Lyttleton  2014 1
Glücksspielmarkt   2
 

Gluckstein, Hannah, 1895-1978 -- See Gluck, 1895-1978


  1
 

Glucocerebrosidase Deficiencies -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
  1
 

Glucocerebrosidase Deficiency -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
  1
 

Glucocerebrosidase Deficiency Disease -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
  1
 

Glucocerebrosidase Deficiency Diseases -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
  1
 

Glucocerebrosidoses -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
  1
 

Glucocerebrosidosis -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement
  1
 

Glucocorticoid-Induced TNFR-Related Protein -- See Also Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY
  1
 

Glucocorticoid Receptor -- See Receptors, Glucocorticoid


Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example
  1
 

Glucocorticoid Receptors -- See Receptors, Glucocorticoid


Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example
  1
  Glucocorticoids -- 2 Related Subjects   2
Glucocorticoids -- adverse effects : Glucocorticoid-induced osteoporosis / volume editors, Andrea Giustina, Alberto Angeli, Ernesto Canalis ; assistant editor, Filippo Manelli  2001 1
Glucocorticoids -- Mechanism of action : The adrenal in toxicology : target organ and modulator of toxicity / edited by Philip W. Harvey  1996 1
Glucocorticoids -- metabolism : The hypothalamic-pituitary-adrenal axis in health and disease : Cushing's syndrome and beyond / Eliza B. Geer, editor  2017 1
 

Glucocorticoids Receptors -- See Receptors, Glucocorticoid


Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example
  1
Glucocorticoids -- Receptors. : The medicinal chemistry of glucocorticoid receptor modulators / Adrian Hobson  2023 1
Glucocorticoids -- therapeutic use   2
Glucocorticoids -- Toxicology : Glucocorticoid-induced osteoporosis / volume editors, Andrea Giustina, Alberto Angeli, Ernesto Canalis ; assistant editor, Filippo Manelli  2001 1
Glucokinase. : Glucokinase and glycemic disease : from basics to novel therapeutics / volume editors, F.M. Matschinsky, M.A. Magnuson  2004 1
Glucokinase -- metabolism : Glucokinase and glycemic disease : from basics to novel therapeutics / volume editors, F.M. Matschinsky, M.A. Magnuson  2004 1
Glucokinase -- Physiologie -- Aufsatzsammlung. : Glucokinase and glycemic disease : from basics to novel therapeutics / volume editors, F.M. Matschinsky, M.A. Magnuson  2004 1
Glucokinase -- therapeutic use : Glucokinase and glycemic disease : from basics to novel therapeutics / volume editors, F.M. Matschinsky, M.A. Magnuson  2004 1
 

Glucolysis -- See Glycolysis


  1
 

Glucophage -- See Metformin


A biguanide hypoglycemic agent used in the treatment of non-insulin-dependent diabetes mellitus not responding to dietary modification. Metformin improves glycemic control by improving insulin sensitivity and decreasing intestinal absorption of glucose. (From Martindale, The Extra Pharmacopoeia, 30th ed, p289)
  1
Glucosamine -- Therapeutic use. : The arthritis cure : the medical miracle that can halt, reverse, and may even cure osteoarthritis / Jason Theodosakis, Brenda Adderly, and Barry Fox  1997 1
 

Glucosaminidase -- See Hexosaminidases


Enzymes that catalyze the hydrolysis of N-acylhexosamine residues in N-acylhexosamides. Hexosaminidases also act on GLUCOSIDES; GALACTOSIDES; and several OLIGOSACCHARIDES
  1
 

Glucosaminidases -- See Hexosaminidases


Enzymes that catalyze the hydrolysis of N-acylhexosamine residues in N-acylhexosamides. Hexosaminidases also act on GLUCOSIDES; GALACTOSIDES; and several OLIGOSACCHARIDES
  1
  Glucose -- 7 Related Subjects   7
Glucose.   10
Glucose -- Analysis.   2
 

Glucose, Blood -- See Blood Glucose


Glucose in blood
  1
 

Glucose, Blood, Self Monitoring -- See Blood Glucose Self-Monitoring


Self evaluation of whole blood glucose levels outside the clinical laboratory. A digital or battery-operated reflectance meter may be used. It has wide application in controlling unstable insulin-dependent diabetes
  1
Glucose -- chemistry : Controlling Maillard pathways to generate flavors / [edited by] Donald S. Mottram, Andrew J. Taylor ; sponsored by the ACS Division of Agricultural and Food Chemistry  2010 1
 

Glucose-Fructose Syrup -- See High Fructose Corn Syrup


Syrup made from corn used widely in foods and beverages as a cheaper alternative sweetener to SUCROSE (common table sugar). It is generated by enzymatic processing of natural corn syrup to produce a liquid most widely composed of 42 or 55% FRUCTOSE, GLUCOSE, and various POLYSACCHARIDES
  1
 

Glucose in blood -- See Blood sugar


  1
  Glucose Intolerance -- 2 Related Subjects   2
Glucose -- Mathematical models : Personalized predictive modeling in Type 1 diabetes / Eleni I. Georga, Dimitrios I. Fotiadis, Stelios K. Tigas  2018 1
 

Glucose Metabolic Disorder -- See Glucose Metabolism Disorders


Pathological conditions in which the BLOOD GLUCOSE cannot be maintained within the normal range, such as in HYPOGLYCEMIA and HYPERGLYCEMIA. Etiology of these disorders varies. Plasma glucose concentration is critical to survival for it is the predominant fuel for the CENTRAL NERVOUS SYSTEM
  1
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