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Orphée et Eurydice -- See Also Orfeo ed Euridice


  1
 

Orpheus and Euridice (1762) -- See Orfeo ed Euridice


  1
 

Orpheus und Eurydike (1762) -- See Orfeo ed Euridice


  1
Gluck, Christoph Willibald, Ritter von, 1714-1787 -- Performances -- France -- Paris -- History -- 19th century : Genealogies of music and memory : Gluck in the 19th-Century Parisian imagination / Mark Everist  2021 1
Gluck, Christoph Willibald, Ritter von, 1714-1787. Rencontre imprévue. : Morality and Viennese opera in the age of Mozart and Beethoven / Martin Nedbal  2017 1
 

Gluck, Cristoforo W., 1714-1787 -- See Gluck, Christoph Willibald, Ritter von, 1714-1787


  1
Gluck, Florence W. : The minor poems / Stephen Hawes ; edited by Florence W. Gluck and Alice B. Morgan  1974 1
Gluck, John Duval, 1878-1951. : The Santa Claus man : the rise and fall of a Jazz Age con man and the invention of Christmas in New York / Alex Palmer  2015 1
Glück, Louise, 1943-2023 : Intimate profiles of five major poets / directed by Mel Stuart for the Academy of American Poets  2009 1
Glück, Louise, 1943-2023 -- Criticism and interpretation   5
Glück, Louise, 1943-2023 -- Musical settings   2
Glück, Louise, 1943-2023. Wild iris : Overheard voices : address and subjectivity in postmodern American poetry / Ann Keniston  2006 1
Glück, Louise, 1943- -- Criticism and interpretation : The poetry of Louise Glück : a thematic introduction / Daniel Morris  2006 1
Glück, Michaël -- Musical settings : Fallen sparrow : [music for electronics and live performers] / Barry Schrader  2006 1
Glück Motiv   5
Gluck, Peter L. : Ten houses : Peter L. Gluck and partners / edited by Oscar Riera Ojeda  1997 1
  Gluck-Piccinni controversy -- 3 Related Subjects   3
Gluck-Piccinni controversy. : Disinformation in mass media : Gluck, Piccinni and the Journal de Paris / Beverly Jerold  2020 1
Glück Politische Institution : Happiness and economics : how the economy and institutions affect well-being / Bruno S. Frey and Alois Stutzer  2002 1
 

Gluck, R. Ch. W. von (Christoph Willibald), Ritter von, 1714-1787 -- See Gluck, Christoph Willibald, Ritter von, 1714-1787


  1
Glück -- Wirtschaft. : Happiness and economics : how the economy and institutions affect well-being / Bruno S. Frey and Alois Stutzer  2002 1
 

Glückel, of Hameln, 1646-1724 -- See Glueckel, of Hameln, 1646-1724


  1
 

Glückel, von Hameln, 1646-1724 -- See Glueckel, of Hameln, 1646-1724


  1
Gluckman, David : Lumkani : rapid detection of fires in informal settlements / Chris van der Hoven, Stephanie Townsend  2018 1
Gluckman, Max, 1911-1975.   2
 

Gluckmann, Herman Max, 1911-1975 -- See Gluckman, Max, 1911-1975


  1
Glückseligkeit : Teaching Happiness and Well-Being in Schools : Learning to Ride Elephants / Ian Morris  2009 1
Glücksspiel   9
Glücksspielindustrie : Intimate economies of development : mobility, sexuality and health in Asia / Chris Lyttleton  2014 1
Glücksspielmarkt   2
 

Gluckstein, Hannah, 1895-1978 -- See Gluck, 1895-1978


  1
 

Glucocerebrosidase Deficiencies -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Glucocerebrosidase Deficiency -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Glucocerebrosidase Deficiency Disease -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Glucocerebrosidase Deficiency Diseases -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Glucocerebrosidoses -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Glucocerebrosidosis -- See Gaucher Disease


An autosomal recessive disorder caused by a deficiency of acid beta-glucosidase (GLUCOSYLCERAMIDASE) leading to intralysosomal accumulation of glycosylceramide mainly in cells of the MONONUCLEAR PHAGOCYTE SYSTEM. The characteristic Gaucher cells, glycosphingolipid-filled HISTIOCYTES, displace normal cells in BONE MARROW and visceral organs causing skeletal deterioration, hepatosplenomegaly, and organ dysfunction. There are several subtypes based on the presence and severity of neurological involvement 		  1
 

Glucocorticoid-Induced TNFR-Related Protein -- See Also Autoimmune Lymphoproliferative Syndrome


Rare congenital lymphoid disorder due to mutations in certain Fas-Fas ligand pathway genes. Known causes include mutations in FAS, TNFSF6, NRAS, CASP8, and CASP10 proteins. Clinical features include LYMPHADENOPATHY; SPLENOMEGALY; and AUTOIMMUNITY 		  1
 

Glucocorticoid Receptor -- See Receptors, Glucocorticoid


Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example 		  1
 

Glucocorticoid Receptors -- See Receptors, Glucocorticoid


Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example 		  1
  Glucocorticoids -- 2 Related Subjects   2
Glucocorticoids -- adverse effects : Glucocorticoid-induced osteoporosis / volume editors, Andrea Giustina, Alberto Angeli, Ernesto Canalis ; assistant editor, Filippo Manelli  2001 1
Glucocorticoids -- Mechanism of action : The adrenal in toxicology : target organ and modulator of toxicity / edited by Philip W. Harvey  1996 1
Glucocorticoids -- metabolism : The hypothalamic-pituitary-adrenal axis in health and disease : Cushing's syndrome and beyond / Eliza B. Geer, editor  2017 1
 

Glucocorticoids Receptors -- See Receptors, Glucocorticoid


Cytoplasmic proteins that specifically bind glucocorticoids and mediate their cellular effects. The glucocorticoid receptor-glucocorticoid complex acts in the nucleus to induce transcription of DNA. Glucocorticoids were named for their actions on blood glucose concentration, but they have equally important effects on protein and fat metabolism. Cortisol is the most important example 		  1
Glucocorticoids -- Receptors. : The medicinal chemistry of glucocorticoid receptor modulators / Adrian Hobson  2023 1
Glucocorticoids -- Therapeutic use   2
Glucocorticoids -- Toxicology : Glucocorticoid-induced osteoporosis / volume editors, Andrea Giustina, Alberto Angeli, Ernesto Canalis ; assistant editor, Filippo Manelli  2001 1
Glucokinase. : Glucokinase and glycemic disease : from basics to novel therapeutics / volume editors, F.M. Matschinsky, M.A. Magnuson  2004 1
Glucokinase -- metabolism : Glucokinase and glycemic disease : from basics to novel therapeutics / volume editors, F.M. Matschinsky, M.A. Magnuson  2004 1
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