Substances used on inanimate objects that destroy harmful microorganisms or inhibit their activity. Disinfectants are classed as complete, destroying SPORES as well as vegetative forms of microorganisms, or incomplete, destroying only vegetative forms of the organisms. They are distinguished from ANTISEPTICS, which are local anti-infective agents used on humans and other animals. (From Hawley's Condensed Chemical Dictionary, 11th ed)
Anti-infective agents -- Therapeutic use -- Congresses : Treating infectious diseases in a microbial world : report of two workshops on novel antimicrobial therapeutics / Committee on New Directions in the Study of Antimicrobial Therapeutics: New Classes of Antimicrobials ; Committee on New Directions in the Study of Antimicrobial Therapeutics: Immunomodulation, Board on Life Sciences, Division on Earth and Life Sciences, National Research Council of the National Academies
Substances used on humans and other animals that destroy harmful microorganisms or inhibit their activity. They are distinguished from DISINFECTANTS, which are used on inanimate objects
Anti-Inflammatory Agents -- administration & dosage : Injection techniques in musculoskeletal medicine : a practical manual for clinicians in primary and secondary care / Stephanie Saunders, Steve Longworth ; foreword by Jonathan Botting
Anti-inflammatory agents that are non-steroidal in nature. In addition to anti-inflammatory actions, they have analgesic, antipyretic, and platelet-inhibitory actions.They act by blocking the synthesis of prostaglandins by inhibiting cyclooxygenase, which converts arachidonic acid to cyclic endoperoxides, precursors of prostaglandins. Inhibition of prostaglandin synthesis accounts for their analgesic, antipyretic, and platelet-inhibitory actions; other mechanisms may contribute to their anti-inflammatory effects
Anti-inflammatory agents that are non-steroidal in nature. In addition to anti-inflammatory actions, they have analgesic, antipyretic, and platelet-inhibitory actions.They act by blocking the synthesis of prostaglandins by inhibiting cyclooxygenase, which converts arachidonic acid to cyclic endoperoxides, precursors of prostaglandins. Inhibition of prostaglandin synthesis accounts for their analgesic, antipyretic, and platelet-inhibitory actions; other mechanisms may contribute to their anti-inflammatory effects
