| Description |
1 online resource (334 p.) |
| Contents |
Front Cover -- TINNITUS AND HYPERACUSIS -- TINNITUS AND HYPERACUSIS: Facts, Theories, and Clinical Implications -- Copyright -- Contents -- Preface -- Abbreviations in the main text -- 1 -- Tinnitus, hyperacusis, and hearing loss -- 1.1 Introduction -- 1.2 Acquired hearing loss -- 1.2.1 Human data -- 1.2.2 Animal data -- 1.3 Loudness recruitment -- 1.3.1 Auditory nerve responses and recruitment -- 1.3.2 Central nervous system and recruitment -- 1.4 Noise-induced temporary threshold shifts -- 1.4.1 Animal data -- 1.4.2 Human findings -- 1.4.2.1 Autopsy material -- 1.4.2.2 Human ABR data |
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1.5 Noise exposure not causing PTS or TTS -- 1.6 Tinnitus pitch, loudness, and hearing loss -- 1.6.1 Tinnitus pitch -- 1.6.2 Tinnitus loudness -- 1.7 Summary -- References -- 2 -- Epidemiology, etiology, and genetics -- 2.1 Prevalence of tinnitus and hyperacusis -- 2.1.1 Overall prevalence -- 2.1.2 Co-occurrence of tinnitus and hyperacusis -- 2.1.3 Tinnitus and auditory hallucinations -- 2.1.4 Co-occurrence of tinnitus and chronic pain -- 2.2 Etiology -- 2.2.1 Hidden hearing loss -- 2.2.2 Acquired hearing loss -- 2.2.3 Blast injury and concussions -- 2.2.3.1 Mechanisms -- 2.2.3.2 Military |
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2.2.3.3 Sports -- 2.2.4 Somatic injury -- 2.2.5 Stress -- 2.2.6 Ménière's disease -- 2.2.7 Tinnitus risk in common diseases -- 2.2.8 Tinnitus and aging -- 2.3 Genetics -- 2.3.1 Hereditary aspects -- 2.3.2 Genome studies -- 2.3.2.1 Candidate gene studies -- 2.3.2.2 Genome-wide association studies -- 2.3.3 Epigenetic factors possibly contributing to tinnitus -- 2.4 Summary -- References -- 3 -- Physiological markers of tinnitus and hyperacusis -- 3.1 Spontaneous neural activity. Noise or information carrier? -- 3.1.1 Auditory nerve fibers -- 3.1.2 Spontaneous firing in auditory cortex |
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3.2 Spike-firing synchrony -- 3.2.1 Sparse cross-correlation in auditory cortex -- 3.2.2 Epidural versus intracortical spike-based receptive fields -- 3.3 Brain rhythms -- 3.3.1 Ultra-slow fluctuations -- 3.3.2 Delta -- 3.3.3 Theta -- 3.3.4 Alpha -- 3.3.5 Beta -- 3.3.6 Gamma -- 3.4 Thalamocortical oscillations -- 3.5 Spike-LFP and EEG/MEG correlation and networks in human cortex -- 3.5.1 Correlation -- 3.5.2 Resting state networks -- 3.6 Auditory-evoked potentials -- 3.6.1 The auditory brainstem response -- 3.6.2 The auditory steady-state response -- 3.6.3 The longer latency obligatory responses |
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3.6.4 The event-related responses -- 3.6.5 Evoked magnetic fields -- 3.7 Summary -- References -- 4 -- Substrates of tinnitus and hyperacusis in the animal auditory system -- 4.1 Spontaneous firing activity in general -- 4.2 Effects of salicylate and noise exposure in auditory pathways -- 4.3 Cell types and afferent pathways -- 4.3.1 The ventral cochlear nucleus -- 4.3.2 The dorsal cochlear nucleus -- 4.4 Hyperacusis -- 4.4.1 Evidence from single units and cortical oscillations -- 4.4.2 Behavioral signs of hyperacusis and tinnitus |
| Notes |
Description based upon print version of record |
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4.4.3 Putative different pathways for igniting hyperacusis and tinnitus |
| Form |
Electronic book
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| ISBN |
9780323985260 |
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0323985262 |
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